±Û·çÆ÷½Ã³×ÀÌÆ®¾Ï¸ð´½(glufosinate ammonium, BASTA¢ç) Áßµ¶ ÈÄ ¹ß»ýÇÑ ³úº´Áõ
Encephalopathy After Glufosinate Ammonium Intoxication
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±èÀçȯ(Kim Jae-Hwan) - °Ç¾ç´ëÇб³ ÀÇ°ú´ëÇÐ ½Å°æ°úÇб³½Ç
À¯Àοì(Yu In-Woo) - °Ç¾ç´ëÇб³ ÀÇ°ú´ëÇÐ ½Å°æ°úÇб³½Ç
±è¿ë´ö(Kim Yong-Duk) - °Ç¾ç´ëÇб³ ÀÇ°ú´ëÇÐ ½Å°æ°úÇб³½Ç
³ª»óÁØ(Na Sang-Jun) - °Ç¾ç´ëÇб³ ÀÇ°ú´ëÇÐ ½Å°æ°úÇб³½Ç
À̱â¿í(Lee Kee-Ook) - °Ç¾ç´ëÇб³ ÀÇ°ú´ëÇÐ ½Å°æ°úÇб³½Ç
À±º¸¶ó(Yoon Bo-Ra) - °Ç¾ç´ëÇб³ ÀÇ°ú´ëÇÐ ½Å°æ°úÇб³½Ç
Abstract
Glufosinate ammonium is the active ingredient in broad-spectrum contact herbicides such as BASTA¨Þ that inhibits the activity of glutamine synthetase, which is necessary for the production of the amino acid glutamine and for ammonia detoxification. Complications of glufosinate ammonium intoxication include gastrointestinal symptoms, loss of consciousness, convulsions, memory impairment, respiratory failure, and cardiovascular instability. We report herein a case of encephalopathy and reversible signal changes in the splenium of the corpus callosum, the bilateral corticospinal tracts, the hippocampi, and the cerebellar peduncles as seen in diffusion-weighted magnetic resonance imaging and fluid-attenuated inversion recovery images following BASTA¨Þ intoxication.
Å°¿öµå
Glufosinate ammonium, Encephalopathy, Intoxication
KMID :
0359720140320020113
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